Here are a few facts about menopause and menstruation that have to do with calcium and magnesium…
Menopause and Menstruation – Calcium
“Between the ages of 15 and 45, women lose at the menstrual period cyclically, during 2 or 3 days of each month, considerable calcium. Calcium requirements of nursing and pregnant women are three times the normal adult requirements. During puberty and at the menopause, glandular overactivity causes excessive losses of calcium, iron and other elements. Similarly, men under a severe mental strain, or those engaged in heavy physical labor, require more calcium to replace losses.
“The glands of internal secretion are all concerned with the proper distribution and utilization of calcium. When the thyroid gland is overactive, experimental animals are known to lose 250% more than the normal amounts of calcium.”
Wow! That’s a lot of calcium to lose – 250%!!
Calcium is considered by many physiologists to be the most important element and is found in the largest amounts in animals. It is vital for the coagulation of blood and milk; and for the formation of bone. The normal beating of the heart is dependent on the relationship between sodium and potassium salts, with calcium. Calcium aids in preserving the alkalinity of the blood and is therefore necessary for the preservation of teeth and bones.
Menopause and Menstruation – Magnesium
“The salts of magnesium aid in the formation of the albumin* of the blood, reduce foreign matter and waste, and maintain the osmotic** pressure of the blood.
The chief storehouse of magnesium is in the bones. It requires the presence of calcium salts for its proper function and is injurious in the absence of calcium. An excess of magnesium in the blood is said to cause greatly increased sugar in the urine. Muscles and nervous tissue contain large amounts of magnesium.”
*Albumin is a protein made by the liver. A serum albumin test measures the amount of this protein in the clear liquid portion of the blood. https://www.nlm.nih.gov/medlineplus/ency/article/003480.htm
With acknowledgement to Dr Royal Lee